Disruption of Slc52a3 gene causes neonatal lethality with riboflavin deficiency in mice

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Hiroki Yoshimatsu; Atsushi Yonezawa; Kaori Yamanishi; Yoshiaki Yao; Kumiko Sugano; Shunsaku Nakagawa; Satoshi Imai; Tomohiro Omura; Takayuki Nakagawa; Ikuko Yano; Satohiro Masuda; Ken-ichi Inui; Kazuo Matsubara;
(2016)
  • Publisher: Nature Publishing Group
  • Journal: Scientific Reports,volume 6 (issn: 2045-2322, eissn: 2045-2322)
  • Related identifiers: doi: 10.1038/srep27557, pmc: PMC4897618
  • Subject: Article
    mesheuropmc: food and beverages | human activities | heterocyclic compounds | digestive, oral, and skin physiology

Homeostasis of riboflavin should be maintained by transporters. Previous in vitro studies have elucidated basic information about riboflavin transporter RFVT3 encoded by SLC52A3 gene. However, the contribution of RFVT3 to the maintenance of riboflavin homeostasis and th... View more
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