E4orf1 Enhances Glucose Uptake Independent of Proximal Insulin Signaling

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Na, Ha-Na; Hegde, Vijay; Dubuisson, Olga; Dhurandhar, Nikhil V.;
(2016)
  • Publisher: Public Library of Science
  • Journal: PLoS ONE,volume 11,issue 8 (issn: 1932-6203, eissn: 1932-6203)
  • Publisher copyright policies & self-archiving
  • Related identifiers: doi: 10.1371/journal.pone.0161275, pmc: PMC4990264
  • Subject: Lipids | Research Article | Insulin | Hormones | Anatomy | Infectious Disease Control | Infectious Diseases | Chemical Compounds | Insulin Signaling | Physical Sciences | Genetics | Biological Tissue | Organic Chemistry | Fats | Endocrine Physiology | Chemistry | Carbohydrates | Glucose | Biology and Life Sciences | Non-coding RNA | RNA | Signal Transduction | Glucose Signaling | Physiology | Adipose Tissue | Medicine | Nucleic acids | Small interfering RNAs | Q | R | Cell Biology | Gene regulation | Science | Biochemistry | Cell Signaling | Monosaccharides | Medicine and Health Sciences | Organic Compounds | Diabetic Endocrinology | Gene expression | Endocrinology

Impaired proximal insulin signaling is often present in diabetes. Hence, approaches to enhance glucose disposal independent of proximal insulin signaling are desirable. Evidence indicates that Adenovirus-derived E4orf1 protein may offer such an approach. This study dete... View more
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