Behavioral and pathophysiological outcomes associated with caffeine consumption and repetitive mild traumatic brain injury (RmTBI) in adolescent rats.

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Yamakawa, Glenn R.; Lengkeek, Connor; Salberg, Sabrina; Spanswick, Simon C.; Mychasiuk, Richelle;
(2017)
  • Publisher: Public Library of Science (PLoS)
  • Journal: PLoS ONE,volume 12,issue 11 (issn: 1932-6203, eissn: 1932-6203)
  • Publisher copyright policies & self-archiving
  • Related identifiers: doi: 10.1371/journal.pone.0187218, pmc: PMC5673214
  • Subject: Animal Models | Research Article | Anatomy | Eukaryota | Prefrontal Cortex | Chemical Compounds | Rodents | Population Groupings | Caffeine | Physical Sciences | Genetics | People and Places | Children | Rats | Animals | Experimental Organism Systems | Neurology | Chemistry | Cellular Types | Biology and Life Sciences | Granule Cells | Adolescents | Alkaloids | Research and Analysis Methods | Medicine | Brain Damage | Brain | Families | Age Groups | Vertebrates | Amniotes | Q | R | Cell Biology | Model Organisms | Mammals | Science | Organisms | Medicine and Health Sciences | Gene Expression | Hypothalamus

Given that caffeine consumption is exponentially rising in adolescents and they are at increased risk for repetitive mild traumatic brain injury (RmTBI), we sought to examine the pathophysiological outcomes associated with early life caffeine consumption and RmTBI. Adol... View more
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