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This replication package contains all code, documentation and metadata for the study "ESG did not immunize stocks during the COVID-19 crisis, but investments in intangible assets did", as published in the Journal of Business Finance & Accounting. Due to contractual limitations, the data are stored in a secure location at Tilburg University and cannot be shared. Additional documentation and metadata can be found in the 'metadata' folder.

Dendrochronological research project

Dendrochronological research project

Quantitative and qualitative data from 51 hosts and 53 refugee guests via TakeCareBnB, all anonimized. The Research Question of this study was: How does a temporal stay with local hosts influence the integration of refugee guests?

Dendrochronological research project

The Wind in our Sails. Places dataset converted to RDF. This deposit contains the resulting RDF triples in separate zip archives as well as scripts used for conversion and linking.

Graph neural networks and other machine learning models offer a promising direction for interpretable machine learning on relational and multimodal data. Until now, however, progress in this area is difficult to gauge. This is primarily due to a limited number of datasets with (a) a high enough number of labeled nodes in the test set for precise measurement of performance, and (b) a rich enough variety of of multimodal information to learn from. Here, we introduce a set of new benchmark tasks for node classification on knowledge graphs. We focus primarily on node classification, since this setting cannot be solved purely by node embedding models, instead requiring the model to pool information from several steps away in the graph. However, the datasets may also be used for link prediction. For each dataset, we provide test and validation sets of at least 1000 instances, with some containing more than 10\;000 instances. Each task can be performed in a purely relational manner, to evaluate the performance of a relational graph model in isolation, or with multimodal information, to evaluate the performance of multimodal relational graph models. All datasets are packaged in a CSV format that is easily consumable in any machine learning environment, together with the original source data in RDF and pre-processing code for full provenance. We provide code for loading the data into \texttt{numpy} and \texttt{pytorch}. We compute performance for several baseline models.

The Wind in our Sails. BGB Dutch Asiatic Shipping dataset converted to RDF. Based on Database Dutch-Asiatic Shipping in the 17th and 18th centuries, 1595-1795 (Huygens ING) http://resources.huygens.knaw.nl/das This deposit contains two parts: DAS and NODAS. For each part this contains the resulting RDF triples in separate zip archives as well as scripts used for conversion and linking.

This record contains raw data related to the article "Impact of Oxidative Stress and Protein S-Glutathionylation in Aortic Valve Sclerosis Patients with Overt Atherosclerosis" Abstract Aortic valve sclerosis (AVSc) is characterized by non-uniform thickening of the leaflets without hemodynamic changes. Endothelial dysfunction, also caused by dysregulation of glutathione homeostasis expressed as ratio between its reduced (GSH) and its oxidised form (GSSG), could represent one of the pathogenic triggers of AVSc. We prospectively enrolled 58 patients with overt atherosclerosis and requiring coronary artery bypass grafting (CABG). The incidence of AVSc in the studied population was 50%. The two groups (No-AVSc and AVSc) had similar clinical characteristics. Pre-operatively, AVSc group showed significantly lower GSH/GSSG ratio than No-AVSc group (p = 0.02). Asymmetric dimethylarginine (ADMA) concentration was significantly higher in AVSc patients compared to No-AVSc patients (p < 0.0001). Explanted sclerotic aortic valves presented a significantly increased protein glutathionylation (Pr-SSG) than No-AVSc ones (p = 0.01). In vitro, inhibition of glutathione reductase caused β-actin glutathionylation, activation of histone 2AX, upregulation of α2 smooth muscle actin (ACTA2), downregulation of platelet and endothelial cell adhesion molecule 1 (PECAM1) and cadherin 5 (CDH5). In this study, we showed for the first time that the dysregulation of glutathione homeostasis is associated with AVSc. We found that Pr-SSG is increased in AVSc leaflets and it could lead to EndMT via DNA damage. Further studies are warranted to elucidate the causal role of Pr-SSG in aortic valve degeneration. This work was supported by the Fondazione Gigi e Pupa Ferrari ONLUS [FPF-14 to P.P. at Centro Cardiologico Monzino] and the Italian Ministry of Health [RC2018-2634599] to P.P at Centro Cardiologico Monzino].